ABC | Volume 113, Nº4, Outubro 2019

Correlação Anatomoclínica Martins et al. Insuficiência cardíaca após infarto sem supradesnivelamento de ST Arq Bras Cardiol. 2019; 113(4):775-782 Figura 8 – Pâncreas vendo-se tanto macroscopicamente (A) quanto em corte histológico (B), coloração pela hematoxilina e eosina, aumento da objetiva 2,5x com lipomatose, substituição das glândulas exócrinas por tecido gorduroso, restando apenas poucas ilhotas de Langerhans (pâncreas endócrino). 1 Figura 8 1. ThygesenK,Alpert JS, JaffeAS,ChaitmanBR,Bax JJ,MorrowDA,etal.Fourth Universal Definition of Myocardial Infarction (2018).J Am Coll Cardiol. 2018,72(18)2231-64. 2. Baron T, Hambraeus K, Sundström J , Erlinge D, Jernberg T, Lindahl B, et al . Type 2myocardial infarction in clinical practice Heart . 2015;101(2):101-6. 3. López-Cuenca A, Gomez-MolinaM, Flores-Blanco PJ, Sánchez-MartínezM, García-Narbon A, De Las Heras-Gómez, et al. Comparison between type-2 and type-1myocardial infarction: clinical features, treatment strategies and outcomes. J Geriatr Cardiol. 2016;13(1):15–22. 4. Saaby L, Poulsen TS, Hosbond S, Larsen TB, Diederichsen ACP, Hallas J, et al. Classification of myocardial infarction: frequency and features of type 2 myocardial infarction. Am J Med. 2013;126(9): 789–97. 5. Chapman AR , Shah ASV, Lee KK, Anand A, Francis O, Adamson P, et al. Long termoutcomes in patients with type 2myocardial infarction andmyocardial injury. Circulation. 2018;137(12):1236-45. 6. Hawatmeh A, Thawabi M, Aggarwal R, Abirami C, Vavilin I, Wasty N, et al. Implications of misclassification of type 2 myocardial infarction on clinical outcomes. Cardiovasc Revasc Med. 2019 Apr 12;Pii:S1553-8389(19)30235-0. 7. Elamm C, Fairweather D, Cooper LT. Pathogenesis and diagnosis of myocarditis. Heart.2012;98(11):835-40. 8. Vos T, Flaxman AD, Naghavi M, Lozano R, Michaud C, Ezzati M, et al.Years lived with disability (YLDs) for 1160 sequelae of 289 diseases and injuries 1990-2010: a systematic analysis for the Global Burden of Disease Study 2010. Lancet. 2012;380(9859):2163-96. 9. MannD, Zipes D, Libby P, BonowR. Braunwald’s Heart Disease: a textbook of cardiovascular medicine, 10th ed, Saunders: Philadelphia; 2017. 10. Montera MW, Mesquita ET, Colafranceschi AS, Oliveira Junior AM, Rabischoffsky A, Ianni BM, et al. I Diretriz Brasileira de Miocardites e Pericardites. Arq Bras Cardiol. 2013;100(4 Suppl 1):1-36. 11. Tschöpe C, Cooper LT, Toree-Amione G, Van Linthout S. Management of myocarditis-relatedcardiomyopathyinadults.CircRes.2019;124(11):1568-83. 12. Johansson BB, Torsvik J, Bjørkhaug L, Vesterhus M, Ragvin A, Tjora E, et al. Diabetes and pancreatic exocrine dysfunction due to mutations in the carboxyl ester lipase gene-maturity onset diabetes of the young (CEL-MODY): a protein misfolding disease. J Biol Chem. 2011;286(40):34593-605. 13. Vesterhus M, Raeder H, Johansson S, Molven A, Njølstad PR. Pancreatic exocrine dysfunction in maturity-onset diabetes of the young type 3. Diabetes Care. 2008;31(2):306-10. 14. Kodvawala A, Ghering AB, Davidson WS, Hui DY. Carboxyl ester lipase expression in macrophages increases cholesteryl ester accumulation and promotes atherosclerosis. J Biol Chem. 2005;280(46):38592-8. 15. Hui DY, Howles PN. Carboxyl ester lipase: structure-function relationship and physiological role in lipoproteinmetabolismand atherosclerosis. J Lipid Res. 2002;43(12):2017-30. Referências Este é um artigo de acesso aberto distribuído sob os termos da licença de atribuição pelo Creative Commons 782