ABC | Volume 110, Nº1, Janeiro 2018
Artigo Original Fischer et al Polimorfismos e extensão da doença coronária Arq Bras Cardiol. 2018; 110(1):16-23 22. YangM,ZhuM,TangL,ZhuH,LuY,XuB,etal.PolymorphismsofTGF β -1and TGFBR2 in relation to coronary artery disease in a Chinese population. Clin Biochem. 2016;49(12):873-8. doi: 10.1016/j.clinbiochem.2016.05.022. 23. Anderson JL, Horne BD, Camp NJ, Muhlestein JB, Hopkins PN, Cannon- Albright LA, et al. Joint effects of common genetic variants from multiple genes and pathways on the risk of premature coronary artery disease. Am Heart J. 2010;160(2):250-256.e3. doi: 10.1016/j.ahj.2010.05.031. 24. Brautbar A, Pompeii LA, Dehghan A, Ngwa JS, Nambi V, Virani SS, et al. A genetic risk score based on direct associations with coronary heart disease improves coronary heart disease risk prediction in the Atherosclerosis Risk in Communities (ARIC), but not in the Rotterdam and Framingham Offspring, Studies. Atherosclerosis. 2012;223(2):421-6. doi: 10.1016/j. atherosclerosis.2012.05.035. 25. Shah S, Casas JP, Gaunt TR, Cooper J, Drenos F, Zabaneh D, et al. Influence of common genetic variation on blood lipid levels, cardiovascular risk, and coronary events in two British prospective cohort studies. Eur Heart J. 2013;34(13):972-81. doi: 10.1093/eurheartj/ehs243. 26. Faludi AA, Izar MC, Saraiva JF, Chacra AP, Bianco HT, Afiune Neto A, et al. Atualizaço da Diretriz Brasileira de dislipidemias e prevenço da aterosclerose – 2017. Arq Bras Cardiol 2017; 109(2Supl.1):1-76. doi: 10.5935/abc.20170121. 27. Sposito AC, Caramelli B, Fonseca FA, Bertolami MC, Afiune Neto A, Souza AD, et al; Sociedade Brasileira de Cardiologia. [IV Brazilian Guideline for DyslipidemiaandAtherosclerosisprevention:DepartmentofAtherosclerosis of Brazilian Society of Cardiology]. Arq Bras Cardiol. 2007;88 Suppl 1:2-19. doi: http://dx.doi.org/10.1590/S0066-782X2007000700002. 28. Chobanian AV, Bakris GL, Black HR, Cushman WC, Green LA, Izzo JL Jr, et al; Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure. National Heart, Lung, and Blood Institute.; National High Blood Pressure Education Program Coordinating Committee. Seventh report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure. Hypertension. 2003;42(6):1206-52. doi: 10.1161/01. HYP.0000107251.49515.c2. 29. Ohkawa H, Ohishi N, Yagi K. Assay for lipid peroxides in animal tissues by thiobarbituricacidreaction.AnalBiochem.1979;95(2):351-8.PMID:36810. 30. Corretti MC, Anderson TJ, Benjamin EJ, Celermajer D, Charbonneau F, Creager MA, et al; International Brachial Artery Reactivity Task Force. Guidelines for the ultrasound assessment of endothelial-dependent flow-mediated vasodilation of the brachial artery: a report of the International Brachial Artery Reactivity Task Force. J Am Coll Cardiol. 2002;39(2):257-65. PMID: 11788217. Erratum in: J Am Coll Cardiol. 2002;39(6):1082. 31. Gensini GG. Amoremeaningful scoring system for determining the severity of coronary heart disease. Am J Cardiol. 1983;51(3):606. PMID: 6823874. 32. Rebhi L, Kchok K, Omezzine A, Kacem S, Rejeb J, Ben HadjMbarek I, et al. Six lipoprotein lipase gene polymorphisms, lipid profile and coronary stenosis in a Tunisian population. Mol Biol Rep. 2012;39(11):9893-901. doi: 10.1007/s11033-012-1856-9 . 33. Corsetti JP, Gansevoort RT, Navis G, Sparks CE, Dullaart RP. LPL polymorphism (D9N) predicts cardiovascular disease risk directly and through interaction with CETP polymorphism (TaqIB) in women with high HDL cholesterol and CRP. Atherosclerosis. 2011;214(2):373-6. doi: 10.1016/j.atherosclerosis.2010.11.029. 34. Ariza MJ, Sánchez-Chaparro MA, Barón FJ, Hornos AM, Calvo-Bonacho E, RiojaJ,etal.AdditiveeffectsofLPL,APOA5andAPOEvariantcombinationson triglyceride levels and hypertriglyceridemia: results of the ICARIA genetic sub- study.BMCMedGenet.2010Apr29;11:66.doi:10.1186/1471-2350-11-66. 35. RigoliL,RaimondoG,DiBenedettoA,RomanoG,PorcelliniA,CampoS,etal. ApolipoproteinAI-CIII-AIVgeneticpolymorphismsandcoronaryheartdisease intype2diabetesmellitus.ActaDiabetol.1995;32(4):251-6.PMID:8750764. 36. SahinS,CeyhanK,Benli I,OzyurtH,NaseriE,TumukluMM,etal.Traditional risk factors and angiotensin-converting enzyme insertion/deletion gene polymorphism in coronary artery disease. GenetMol Res. 2015;14(1):2063- 8. doi: 10.4238/2015.March.20.16. 37. TangWH, Hartiala J, Fan Y,Wu Y, Stewart AF, Erdmann J, et al; CARDIoGRAM Consortium. Clinical and genetic association of serum paraoxonase and arylesterase activities with cardiovascular risk. Arterioscler Thromb Vasc Biol. 2012;32(11):2803-12. doi: 10.1161/ATVBAHA.112.253930. 38. Hou X, Chen X, Shi J. Genetic polymorphism of MTHFR C677T and premature coronary artery disease susceptibility: a meta-analysis. Gene. 2015;565(1):39-44. doi: 10.1016/j.gene.2015.03.062. 39. HelfensteinT,FonsecaFA,RelvasWG,SantosAO,DabelaML,MatheusSC,et al.Prevalenceofmyocardialinfarctionisrelatedtohyperhomocysteinemiabut not influenced by C677Tmethylenetetrahydrofolate reductase andA2756G methionine synthase polymorphisms in diabetic and non-diabetic subjects. Clin ChimActa. 2005;355(1-2):165-72. doi: 10.1016/j.cccn.2004.12.002 . 40. Mahmoodi K, Nasehi L, Karami E, Soltanpour MS. Association of nitric oxide levels and endothelial nitric oxide synthase G894T polymorphism with coronary artery disease in the Iranian population. Vasc Specialist Int. 2016;32(3):105-12. doi: 10.5758/vsi.2016.32.3.105. 41. LiuD,JiangZ,DaiL,ZhangX,YanC,HanY.Associationbetweenthe-786T>C 1polymorphism in the promoter region of endothelial nitric oxide synthase (eNOS) and risk of coronary artery disease: a systematic review and meta- analysis. Gene. 2014;545(1):175-83. doi: 10.1016/j.gene.2013.09.099. Este é um artigo de acesso aberto distribuído sob os termos da licença de atribuição pelo Creative Commons 23
Made with FlippingBook
RkJQdWJsaXNoZXIy MjM4Mjg=