ABC | Volume 110, Nº4, Abril 2018

Artigo Original Wang et al Contagem de monócitos e carga trombótica Arq Bras Cardiol. 2018; 110(4):333-338 1. Taleb S. Inflammation in atherosclerosis. Arch Cardiovasc Dis. 2016;109(12):708-15. doi: 10.1016/j.acvd.2016.04.002. 2. Dutta P, Nahrendorf M. Monocytes in myocardial infarction. Arterioscler Thromb Vasc Biol. 2015;35(5):1066-70. doi: 10.1161/ ATVBAHA.114.304652. 3. Wang Z, Ren L, Liu N, Lei L, Ye H, Peng J. Association of monocyte count on admissionwith angiographic no-reflowafter primary percutaneous coronary intervention in patients with ST-segment elevation myocardial infarction. Kardiol Pol. 2016;74(10):1160-1166. doi: 10.5603/KP.a2016.0065. 4. Gibson CM, de Lemos JA, Murphy SA, Marble SJ, McCabe CH, Cannon CP, et al. Combination therapy with abciximab reduces angiographically evident thrombus in acute myocardial infarction: a TIMI 14 substudy. Circulation. 2001;103(21):2550-4. doi: https://doi.org/10.1161/01. CIR.103.21.2550. 5. Sianos G, Papafaklis MI, Serruys PW. Angiographic thrombus burden classification in patients with ST-segment elevation myocardial infarction treated with percutaneous coronary intervention. J Invasive Cardiol. 2010;22(10 Suppl B):6B-14B. 6. Higuma T, Soeda T, Yamada M, Yokota T, Yokoyama H, Izumiyama K, et al. Does residual thrombus after aspiration thrombectomy affect the outcome of primary PCI in patients with ST-segment elevation myocardial infarction?:Anopticalcoherencetomographystudy. JACCCardiovasc Interv. 2016;9(19):2002-11. doi: 10.1016/j.jcin.2016.06.050. 7. Piccolo R, Galasso G, Iversen AZ, Eitel I, Dominguez-Rodriguez A, Gu YL, et al.Effects of baseline coronary occlusion and diabetes mellitus in patients with ST-segment elevation myocardial infarction undergoing primary percutaneous coronary intervention. Am J Cardiol. 2014;114(8):1145-50. doi: 10.1016/j.amjcard.2014.07.030 8. Lee WC, Chen HC, Fang HY, Hsueh SK, Chen CJ, Yang CH, et al. Comparisonofdifferentstrategies foracuteST-segmentelevationmyocardial infarction with high thrombus burden in clinical practice: Symptom-free outcome at one year. Heart Lung. 2015;44(6):487-93. doi: 10.1016/j. hrtlng.2015.08.003. 9. Teng N, Maghzal GJ, Talib J, Rashid I, Lau AK, Stocker R. The roles of myeloperoxidase in coronary artery disease and its potential implication in plaque rupture. Redox Rep. 2017;22(2):51-73. doi: 10.1080/13510002.2016.1256119. 10. Chistiakov DA, Orekhov AN, Bobryshev YV. Contribution of neovascularization and intraplaque haemorrhage to atherosclerotic plaque progression and instability. Acta Physiol (Oxf). 2015;213(3):539-53. doi: 10.1111/apha.12438. 11. Cimmino G, Loffredo FS, Morello A, D’Elia S, De Palma R, Cirillo P, et al. Immune-inflammatory activation in acute coronary syndromes: a look into the heart of unstable coronary plaque. Curr Cardiol Rev. 2017;13(2):1-8. doi: 10.2174/1573403X12666161014093812. 12. Hisada Y, Alexander W, Kasthuri R, Voorhees P, Mobarrez F, Taylor A, et al. Measurement of microparticle tissue factor activity in clinical samples: A summary of two tissue factor-dependent FXa generation assays. Thromb Res. 2016 Mar;139:90-7. doi: 10.1016/j.thromres.2016.01.011. 13. Palmerini T, Tomasi L, Barozzi C, Della Riva D, Mariani A, Taglieri N, et al. Detection of tissue factor antigen and coagulation activity in coronary artery thrombi isolated from patients with ST-segment elevation acute myocardial infarction. PLoS One. 2013;8(12):e81501. doi: 10.1371/ journal.pone.0081501. 14. Zhang SZ, Jin YP, Qin GM, Wang JH. Association of platelet-monocyte aggregates with platelet activation, systemic inflammation, and myocardial injury in patients with non-ST elevation acute coronary syndromes. Clin Cardiol. 2007;30(1):26-31. doi: 10.1002/clc.2. 15. Ren F, MuN, Zhang X, Tan J, Li L, Zhang C, et al. Increased platelet-leukocyte aggregates are associated with myocardial no-reflow in patients with ST elevation myocardial infarction. Am J Med Sci. 2016;352(3):261-6. doi: 10.1016/j.amjms.2016.05.034. 16. AlemanMM, Gardiner C, Harrison P,Wolberg AS. Differential contributions of monocyte- and platelet-derived microparticles towards thrombin generation and fibrin formation and stability. J Thromb Haemost. 2011;9(11):2251-61. doi: 10.1111/j.1538-7836.2011.04488.x. 17. MachF,SchönbeckU,Bonnefoy JY,Pober JS,LibbyP.Activationofmonocyte/ macrophage functions related to acute atheroma complication by ligation of CD40: induction of collagenase, stromelysin, and tissue factor. Circulation. 1997;96(2):396-9. doi: https://doi.org/10.1161/01.CIR.96.2.396. 18. Rittersma SZ, van derWal AC, Koch KT, Piek JJ, Henriques JP, Mulder KJ, et al. Plaque instability frequently occurs days or weeks before occlusive coronary thrombosis: a pathological thrombectomy study in primary percutaneous coronary intervention. Circulation. 2005;111(9):1160-5. doi: 10.1161/01. CIR.0000157141.00778.AC . Referências Este é um artigo de acesso aberto distribuído sob os termos da licença de atribuição pelo Creative Commons 338