ABC | Volume 113, Nº4, October 2019

Original Article Gil et al. LV function in HCM by Strain technique Arq Bras Cardiol. 2019; 113(4):677-684 Figure 3 – Global longitudinal strain systolic peak curves in the four-chamber apical section (left) and the parametric image of the left ventricle in the bulls-eye (right) in a patient with HCM and hypertension. Table 1 – Characteristics of HCM patients in the different groups Variables Normotensive patients (n = 31) Hypertensive patients (n = 14) p Age (years) 40.16 ± 12.73 56.14 ± 13.87 0.0001 Male sex 15 (48%) 7 (50%) 0.9323 BMI (kg/m 2 ) 25.6 ± 3.97 29.2 ± 2.93 0.0045 SBP (mmHg) 113 ± 12 128 ± 12 0.0004 DBP (mmHg) 71 ± 9 81 ± 9 0.0027 Functional Class (NYHA) 0.1110 I 12 (38.7%) 2 (14.3%) II 19 (61.29%) 11 (78.57%) III 0 (0%) 1 (7.14%) Hypertrophy type 0.1492 I 5 (16.1%) 2 (14.3%) II 12 (38.7%) 2 (14.3%) III 12 (38.7%) 6 (42.9%) IV 2 (6.5%) 4 (28.5%) LVOT obstruction 9 (29%) 6 (43%) 0.5133 Medications Beta-blocker 22 (70%) 12 (86%) 0.4578 ACEI 2 (6.45%) 4 (28.57%) 0.0651 ARB 1 (3.23%) 11 (78.57%) < 0.0001 Calcium antagonist 2 (6.45%) 5 (35.71%) 0.0226 Nitrate 1 (3.23%) 1 (7.14%) 0.0503 Hydralazine 0 (0%) 1 (7.14%) 0.3111 Diuretics 0 (0%) 8 (57.14%) < 0.0001 Values expressed as mean±standard deviation or proportion, as indicated. BMI: body mass index; SBP: systolic blood pressure; DBP: diastolic blood pressure; NYHA: New York Heart Association; ACEI: angiotensin converting enzyme inhibitor, ARB: angiotensin receptor blocker. 680