ABC | Volume 112, Nº6, June 2019

Anatomopathological Correlation Arq Bras Cardiol. 2019; 112(6):793-802 Issa and Benvenuti Pulmonary infiltrate and left ventricular apex obliteration in a young man 1. Anderson Jr FA, Spencer FA. Risk factors for venous thromboembolism. Circulation. 2003;107(23 Suppl 1):I9-I16. 2. Konstantinides SV, Torbicki A, Agnelli G, Danchin N, Fitzmaurice D, Galiè N, et al. 2014 ESC guidelines on the diagnosis and management of acute pulmonary embolism. Eur Heart J. 2014;35(43):3033-69, 3069a-3069k. 3. Corrado D, Basso C, Schiavon M, Thiene G. Screening for hypertrophic cardiomyopathy in young athletes. N Engl J Med. 1998;339(6):364-9. 4. Maron MS, Maron BJ, Harrigan C, Buros J, Gibson CM, Olivotto I, et al. Hypertrophic cardiomyopathy phenotype revisited after 50 years with cardiovascular magnetic resonance. J Am Coll Cardiol. 2009;54(3):220-8. 5. Klues HG, Schiffers A, Maron BJ. Phenotypic spectrum and patterns of left ventricular hypertrophy in hypertrophic cardiomyopathy: morphologic observations and significance as assessed by two-dimensional echocardiography in 600 patients. J AmColl Cardiol. 1995;26(7):1699-708. 6. Arad M, Penas-Lado M, Monserrat L, Maron BJ, Sherrid M, Ho CY, et al. Gene mutations in apical hypertrophic cardiomyopathy. Circulation. 2005;112(18):2805-11. 7. Beaton A, Mocumbi AO. Diagnosis and management of endomyocardial fibrosis. Cardiol Clin. 2017;35(1):87-98. 8. Nunes MC, Guimarães Jr MH, Diamantino AC, Gelape CL, Ferrari TC. Cardiac Manifestations of parasitic diseases. Heart. 2017;103(9):651-8. 9. Alter P, Maisch B. Endomyocardial fibrosis in Churg-Strauss syndrome assessed by cardiac magnetic resonance imaging. Int J Cardiol. 2006;108(1):112-3. 10. Hellmark T, Segelmark M. Diagnosis and classification of Goodpasture’s disease (anti-GBM). J Autoimmun. 2014 Feb-Mar;48-49:108-12. 11. Cahill TJ, Prendergast BD. Infective endocarditis. Lancet. 2016;387 (10021):882-93. 12. Siciliano RF, Mansur AJ, Castelli JB, Arias V, Grinberg M, Levison ME, et al. Community-acquired culture-negative endocarditis: clinical characteristics and risk factors for mortality. Int J Infect Dis. 2014 Aug;25:191-5. 13. Issa VS, Dinardi LF, Pereira TV, Almeida LK, Barbosa TS, Benvenutti LA, et al. Diagnostic discrepancies in clinical practice: An autopsy study in patients with heart failure. Medicine (Baltimore). 2017;96(4):e5978. 14. Jobs A, Simon R, Waha S, Rogacev K, Katalinic A, Babaev V, et al. Pneumonia and Inflammation in acute decompensated heart failure: a registry-based analysis of 1939 patients. Eur Heart J Acute Cardiovasc Care. 2018;7(4):362-70 15. Elliott P, McKenna W. Hypertrophic cardiomyopathy. Lancet. 2004;363(9424):1881-91. 16. Iglezias SD, Benvenuti LA, Calabrese F, Salemi VM, Silva AM, Carturan E, et al. Endomyocardial fibrosis: pathological andmolecular findings of surgically resectedventricularendomyocardium.VirchowsArch.2008;453(3):233-41. 17. Salemi VM, Iglezias SD, Benvenuti LA, Filho JC, Rochitte CE, Shiozaki AA, et al. An unusual association of endomyocardial fibrosis and hypertrophic cardiomyopathy in a patient with heart failure. Cardiovasc Pathol. 2012;21(2):e23-5. 18. Thiene G, Basso C, Calabrese F, Angelini A, Valente M. Twenty years of progresss and beckoning frontiers in cardiovascular pathology: cardiomyopathies. Cardiovasc Pathol. 2005;14(4):165-9. 19. Arbustini E, Narula N, Dec GW, Reddy KS, Greenberg B, Kushwaha S, et al. TheMOGE (S) classification for a phenotype-genotype nomenclature of cardiomyopathy. J Am Coll Cardiol. 2013;62(22):2046-72. References This is an open-access article distributed under the terms of the Creative Commons Attribution License cavity thrombosis at different development stages, both the left ventricular inflow and outflow tracts were affected, which is not described in endomyocardial fibrosis and typically affects only the ventricular apex and inflow tract. 16 It is noteworthy that we have previously reported the simultaneous occurrence of both cardiomyopathies, with typical findings, in a patient who underwent surgical resection of endomyocardial fibrosis and subsequently died. 17 The present case illustrates the difficulty to classify the cardiomyopathy into one of the four traditional basic types, namely: dilated, hypertrophic, restrictive, and arrhythmogenic, 18 and there is a current trend towards a purely descriptive classification, i.e., the MOGE(S) classification. 19 Regarding the suspected Goodpasture syndrome, the necropsy did not show lesions in the renal glomeruli or evidence of vasculitis in the lungs or other organs, and the alveolar pulmonary hemorrhage can be explained by the heart failure and terminal cardiogenic shock, which is the cause of death. Therefore, there are no anatomopathological elements to allow the diagnosis of Goodpasture syndrome, which is in line with the results of the performed complementary tests. The thyroid atrophy corresponds to the sequela of the radioactive iodine treatment of the patient’s hyperthyroidism. (Dr. Luiz Alberto Benvenuti) 802