ABC | Volume 110, Nº1, January 2018

Case Report Avila et al Kawasaki disease and Pregnancy Arq Bras Cardiol. 2018; 110(1):97-100 Figure 1 – (Aand B) Echocardiography: ejection fraction 68%; left atrium 35 mm; septum 8mm; posterior wall 8mm; left ventricular diastolic diameter 45mm; left ventricular systolic diameter 30 mm; PSAP 40 mmHg. Dilatation of left coronary artery (7 mm). Left ventricle with preserved systolic function and myocardial thickness, with no changes in segmental wall motion. (C and D) Cardiac catheterization (10/2013): coronary artery ectasia. Dominant coronary with 50% proximal, eccentric tubular lesion and coronary thrombosis; left coronary artery trunk with aneurysmatic dilatation at the distal third; anterior descending artery with ectasia at the proximal third, without obstructive lesions. Circumflex artery with proximal ectasia, without obstructive lesions. Figure 2 – Coronary computed tomography angiography showed aneurysm at the distal third of the left coronary artery trunk (9mm) and anterior descending artery ostium (9.5 mm); circumflex artery with ectasia at the ostium (3.7 mm); marginal branch 2 with aneurysm at the distal segment; right coronary artery with saccular aneurysm (9.8 mm at the greatest diameter), mural thrombus and small regions of calcification. Total score of 23.81 (Agaston) and 38.59 (Volume). MT: main trunk of the left coronary artery; DA: anterior descending artery; DB: diagonal branch; CA: circumflex artery; M1: left marginal artery; M2: second marginal branch of circumflex artery; RCA: right coronary artery; LA: left atrium; Ao: aorta at this time, enoxaparin was suspended and the patient was discharged. At the clinical visit 60 days thereafter, the patient was asymptomatic, breast feeding and using warfarin (INR = 2) and ASA (100 mg/d). Discussion In the present case, strategies for prevention of complications of giant CAAs secondary to KD and acute infarction were successful in terms of maternal-fetal health. 98